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NTHi: Nontypeable H influenzae Pathogenesis Inflammation Escape bind with the Sap mediated import of receptor on the AMPS PART 2 airway epithelial cell reduce periplasmic and inner Pathogencity v release of membrane accumulation of And proinflammatory these host defense peptides Immunity ytokines and chemokines √ dependent of the NF2KB pathway and independent of the|亟 NF2KB pathway 界R Medical ATP ADP A Microbiology degradationPART 2 Pathogenicity And Immunity Medical Microbiology Inflammation :  bind with the receptor on the airway epithelial cell  release of proinflammatory cytokines and chemokines.  dependent of the NF2κB pathway and independent of the NF2κB pathway Escape:  Sap mediated import of AMPs  reduce periplasmic and inner membrane accumulation of these host defense peptides. NTHi: Nontypeable H. influenzae Pathogenesis
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