Overview Clinical syndrome Slowing down of metabolism Slowing of growth and mental retardation in infants and children Generalized slowing down of organism Deposition of glycosaminoglycans in skin and muscle Reversible with therapy

Pathological changes -- glomerular injury Clinical manifestations --proteinuria / hematuria A group of diseases Complicated causes & mechanisms Various clinical manifestations Different prognosis Multiple treatment

Cirrhosis---definition chronic, progressed, diffuse hepatocellular injury fibrosis nodular regeneration

How to Teach Internal Medicine 2005.2-7: Lecture (Bilingual teaching) 2005.9-2006.1 Bedside teaching: rotation for 6 weeks, including Respiratory system, Cardiovascular system, Gastrointestinal system, Kidney and urinary system, Endocrinology, Hematology, etc

Key Words Respiratory failure Acute respiratory distress syndrome(ARDS) dyspnea Hypoxemia hypercapnia Respiratory Support

Type 1 (beta-cell destruction, usually leading to absolute insulin deficiency) Autoimmune Idiopathic Type 2 – (may range from predominantly insulin resistance with relative

Cardiovascular disease Cerebrovascular diseases Lower respiratory infections Diarrhea Perinatal disease COPD Tuberculosis Morbilli

Chapter three Chronic Obstructive Pulmonary Diseases (COPD) Chronic Bronchitis Obstructive Pulmonary Emphysema

Background Carbon monoxide (CO) is a colorless, odorless gas produced by incomplete combustion of carbonaceous material CO is formed as a by-product of burning organic compounds

Background Sedative-hypnotics are a group of drugs that cause CNS depression. Benzodiazepines (BZD) barbiturates nonbarbiturate nonbenzodiazepine sedative￾hypnotics (NBNB) the most commonly used agents