ANTITHROMBOTIC PROPERTIES Inhibition of platelet aggregation: PGL NO, ADPase Anticoagulant -binding and inhibition of thrombin: Antithrombin acceleration by heparin-like molecules, thrombomodulin activation of protein C/S, a2-macroglobulin. Fibrinlysis: Tissue plasminogen activator (tPA)

一、概述 二、病因学 三、病理学 四、临床表现及诊断 五、治疗 六、预后

温州医科大学：《病理学》课程教学资源（护理学专业，练习资料，人卫九版，含答案）

温州医科大学：临床医学专业《病理学》课程教学资源（练习资料，人卫九版，含答案）

温州医科大学：《病理学》课程教学资源（练习题，人卫专科八版，含答案）

慢性肉芽肿性疾病 (chronic guanulomatous diseasse) 一、X连锁/常染色体隐性遗传性疾病 二、 NADPH氧化酶缺乏 三、过氧化氢酶阳性菌感染

Motional pathway of embolus: Embolus from left heart cavity or arterial system Embolus from right heart cavity or venous system Embolus from portal veins Paradoxical embolism Retrograde ethrombus

CELLULAR ADAPTATION Excessive physiologic stresses. Some pathologic stimuli. A new, but altered state preserving the viability of the cell

Definition of pathology Pathology is to study diseases by scientific methods. Disease may be defined as an abnormal alteration of structure or function in any part of the body

FATTY CHANGE Excessive entry of free fatty acids into the liver (starvation, corticosteroid therapy). Enhanced fatty acid synthesis. Decreased fatty acid oxidation. Increased esterification of fatty acid to triglycerides (alcohol). Decreased apoprotein synthesis (CCl). Impaired lipoprotein secretion from the liver (alcohol)