山东大学：《医学生理学 Medical Physiology》课程教学资源（PPT课件讲稿，人体生理学 Human Physiology）Chapter 4 Cardiovascular Physiology section 2 Electrical Properties of the Cardiac Cell

山东大学：《医学生理学 Medical Physiology》课程教学资源（PPT课件讲稿，人体生理学 Human Physiology）Chapter 4 Cardiovascular Physiology Section 1 The Heart as a Pump

I Signal Transmission Through the Neuromuscular Junction II Microstructure of Skeletal Muscle Begin cycle with myosin already bound to actin IV Factors that Affect the Efficiency of Muscle Contraction

Section 1 Composition and Properties of the Blood Section 2 Hemostasis, Blood Coagulation and Fibrinolysis Section 3 Blood Group and Transfusion Agglutination（凝集）, hemolysis （溶血）and the transfusion reaction

I. Membrane Resting Potential II Action Potential III Initiation of Action Potential Electrophysiological Method to Record Membrane Potential I Voltage Clamp IV Local Response V Excitation and Excitability of the Tissue

I Review Structure of the Cell Membrane II Transport Through the Cell Membrane Tetrodotoxin (TTX) selectively block the voltage-gated Na+ channel ➢ Simple diffusion ➢ Ion channel ➢ Water channel Water transportation through the membrane 3 Active transport 4. Bulk Transport (Endocytosis and Excytosis)

山东大学：《医学生理学 Medical Physiology》课程教学资源（PPT课件讲稿，人体生理学 Human Physiology）Chapter 1 Introduction to Physiology（主讲：刘传勇）

§1 Blood coagulation Blood coagulatlon system Blood platelet §2 Anticoagulation 1. Cell reticuloendothelial system 2. Body fluid (1) antithrombin Ⅲ (AT-Ⅲ,liver): (2) Protein C (3) tissue factor pathway inhibitor (TFPI) §3 Fibrinolysis • 组成：纤维蛋白溶酶原、纤溶酶、纤溶酶原激活物与纤溶抑制物。 • 作用：使生理止血过程中所产生的局部或一过性的纤维蛋白凝块能随时溶解，从而防止血栓形成，保证血流通畅；参与组织修复、血管再生。 §4 Vascular endothelial cells VEC正常时不表达TF； VEC可生成PGI2、NO及ADP酶等物质，扩张血管、抑制血小板的活化、聚集等； VEC可产生或促进tPA、uPA等纤溶酶原激活物，促进纤溶过程； VEC的抗凝作用 • 产生TFPI、表达TM、肝素样物质

第一节 概述 第二节 心力衰竭的病因和诱因 第三节 心力衰竭的分类 第四节 心力衰竭的发病机制 第五节 心功能不全的代偿机制 第六节 心脏泵血功能障碍对机体的主要影响 第七节 心力衰竭防治的病理生理基础

一、血细胞的生成部位 卵黄囊→肝、脾→骨髓→不规则骨胚胎早期第2个月第个4月18岁以后